Afterwards, she underwent blood perfusion and was given methylprednisolone intravenously. On day 1 of poisoning, she was immediately given gastric lavage and catharsis. The urine semiquantitative method result was dark blue color. The arterial blood gas analysis (ABG) showed PO 2/FiO 2 of 381.0 mmHg and Lac of 2.9 mmol/L ( Fig. The patient was presented to our ER 2 hr after poisoning, with SpO 2 of 98%. This case will help clinicians gain a better understanding of the long-term outcomes of patients who experienced PQ poisoning, especially the effects of poisoning on airway function.Ī 62-year-old female farmer without chronic respiratory disease or allergy history intentionally drank approximately 10 g of paraquat on June 16, 2015. The presenting symptoms were repeated asthma attacks with severe ventilatory disorder, but chest computed tomography (CT) did not detect interstitial fibrosis or other structural disorders.
In our emergency room (ER), a patient who experienced PQ poisoning with no underlying pulmonary disease was followed up for more than 5 years to evaluate long-term changes in pulmonary function and lung imaging findings. However, few studies have reported pulmonary function changes in patients who have survived for a longer time, especially those who have lived for more than 5 years after poisoning. In survivors, lung imaging and respiratory functions revealed partial recovery 2–3 years after poisoning ( Lee et al., 2009).
Most patients die from respiratory failure. Patients who experienced acute paraquat (PQ) poisoning always present with acute respiratory distress syndrome in the early stage that gradually progresses to pulmonary fibrosis in the subacute stage.
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